An engineered cell line lacking OGG1 and MUTYH glycosylases implicates the accumulation of genomic 8-oxoguanine as the basis for paraquat mutagenicity. Tajai, P., Fedeles, B. I., Suriyo, T., Navasumrit, P., Kanitwithayanun, J., Essigmann, J. M., &Satayavivad, J. (2018). Free Radical Biology and Medicine, 116, 64-72.

28 Dec 2018
0
0

An engineered cell line lacking OGG1 and MUTYH glycosylases implicates the accumulation of genomic 8-oxoguanine as the basis for paraquat mutagenicity.
Tajai, P., Fedeles, B. I., Suriyo, T., Navasumrit, P., Kanitwithayanun, J., Essigmann, J. M., &Satayavivad, J. (2018).
Free Radical Biology and Medicine, 116, 64-72.

Tags

Share this

Related Posts

Hung, M. H., Lee, J. S., Ma, C., Diggs, L. P., Heinrich, S., Chang, C. W., . . . Wang, X. W. (2021). Tumor methionine metabolism drives T-cell exhaustion in hepatocellular carcinoma. Nat Commun, 12(1), 1455. doi:10.1038/s41467-021-21804-1
05 Mar 2021
0

Hung, M. H., Lee, J. S., Ma, C., Diggs, L. P., Heinrich, S., Chang, C. W., . . . Wang, X. W. (2021). Tumor methionine metabolism drives T-cell exhaustion in hepatocellular carcinoma. Nat Commun, 12(1), 1455. doi:10.1038/s41467-021-21804-1

Hung, M. H., Lee, J. S., Ma, C., Diggs, L. P., Heinrich, S., Chang, C. W., . . . Wang, X. W. (2021). Tumor methionine metabolism drives T-cell exhaustion in hepatocellular carcinoma. Nat Commun, 12(1), 1455. doi:10.1038/s41467-021-21804-1
View More

Recent Posts